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11th Annual Congress on Immunology & Immunotechnology, will be organized around the theme ““Disseminating the next generation facets of Immunology and Immunotechnology” ”

Immunology Congress 2018 is comprised of 26 tracks and 323 sessions designed to offer comprehensive sessions that address current issues in Immunology Congress 2018.

Submit your abstract to any of the mentioned tracks. All related abstracts are accepted.

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Immunogenomics originally was framed by research supporting the hypothesis that cancer mutations generated novel peptides seen as ‘‘non-self’’ by the immune system. ‘Neoantigens’ has been facilitated by the combination of specialized computational analyses, new sequencing technologies, and HLA binding predictions that evaluate somatic alterations in a cancer genome and interpret their ability to produce an immune-stimulatory peptide. The resulting information can characterize a tumor’s Neoantigens load, its cadre of infiltrating immune cell types, the T or B cell receptor repertoire, and direct the design of a personalized therapeutic.

  • Track 1-1Immune deficiency diseases
  • Track 1-2Immunological disorders
  • Track 1-3Innate and Adaptive Immune system
  • Track 1-4Neoantigens
  • Track 1-5Immune Repertoire Profiling
  • Track 1-6Distribution of Tumor Infiltrating Lymphocytes
  • Track 1-7Application of Immunogenomics data for disease association studies

Immunotoxicology is the investigation of immune dysfunction resulting from exposure of an organism to a xenobiotic. Immunotoxicology is moderately new interdisciplinary logical field concentrated on recognizable proof and investigation of the compound and, in a more extensive sense, additionally physical and organic elements of nature which can bring about undesirable and normally accidental immunomodulation. The immune dysfunction may take the form of immunosuppression, sensitivityautoimmunity, and incendiary based illnesses. Insusceptible System assumes a basic part in host imperviousness to sickness and additionally in typical homeostasis of a living being; recognizable proof of immunotoxic hazard is huge in the assurance of human, creature and wildlife health. In addition, immunotoxicology likewise researches the properties of new immunotherapeutic pharmacological items arranged by means of recombinant DNA methods (interleukins, interferons, development variables, hostile to irritation drugs, neuroendocrine hormones, neuropeptides with respect to their immunotoxic potential and security of their utilization.

A biomarker is a trademark that can be unbiased measured as a marker of typical organic procedures, pathogenic procedures or a pharmacological reaction to a restorative mediation. These are utilized for some reasons including malady conclusion and guess, forecast and appraisal of treatment reaction. Biomarkers can be trademark natural properties or particles that can be recognized and measured in parts of the body like the blood or tissue. They may show either ordinary or infected procedure in the body. Biomarkers can be particular cells, particles, or genes, gene items, catalysts, or hormones.

  • Track 2-1Genomic Or DNA Biomarkers
  • Track 2-2Safety Considerations
  • Track 2-3Adverse Immunostimulation
  • Track 2-4Immunosuppression
  • Track 2-5Hypersensitivity
  • Track 2-6Immunogenicity
  • Track 2-7Drug Toxicology
  • Track 2-8Genetic Toxicology
  • Track 2-9Developmental Immunotoxicology
  • Track 2-10Evaluating Immune Biomarkers
  • Track 2-11Cancer Biomarkers
  • Track 2-12Biomarkers in Medicine
  • Track 2-13Proteomic Biomarkers
  • Track 2-14Drug-Related Biomarkers
  • Track 2-15Transcriptomic Biomarkers
  • Track 2-16Genomic biomarkers in cancer Drug discovery
  • Track 2-17Other Related Topics

Immunology is worried about the utilization of immunological responses for the determination, avoidance, and treatment of various infections. It is firmly identified with therapeutic and veterinary microbiology, the study of disease transmission, physiology and Pathophysiology, organic chemistry, and endocrinology. Viral Immunology and the immunology of parasitic infections are autonomous branches of commonsense immunology. Immunology concentrates the antigenic creation of microorganisms, attributes of the safety procedures in different sorts of contaminations, and nonspecific types of Imperviousness to the causative operators of irresistible infections. Investigation of the immunological procedures and the immunological reproduction of the living being created by Non-infectious Antigens of the exogenous and endogenous starting point and the advancement of strategies for controlling unfavorably susceptible sicknesses are winding up noticeably progressively imperative. Different branches of clinical immunology are likewise growing seriously. These incorporate radiation immunology, which concentrates the disturbance of immunological reactivity by illumination, and Immunohematology, which researches the antigenic piece of platelets and the causes and instrument of advancement of immunological harm to the circulatory framework. Immunology is creating techniques for Immunoprophylaxis, Immunotherapy, and Immunodiagnostics.

Immunologic Research speaks to an interesting medium for the introduction, translation, and elucidation of complex logical information. Data is displayed as interpretive combination surveys, unique research articles, symposia, publications, and hypothetical expositions. The extent of scope stretches out to cell immunology, immunogenetics, sub-atomic and auxiliary immunology, immunoregulation and autoimmunity, immunopathology, tumor immunology, have safeguard and microbial resistance, including viral immunology, immunohematology, mucosal invulnerability, supplement, transplantation immunology, clinical immunology, neuroimmunology, immunoendocrinology, immunotoxicology, translational immunology, and history of immunology.

  • Track 3-1Antibody Engineering & Technology
  • Track 3-2International Reviews of Immunology
  • Track 3-3Funding Opportunities in Immunology
  • Track 3-4Novel Approaches in Immunology
  • Track 3-5Technology Development and Applications
  • Track 3-6Nano-Immunotechnology
  • Track 3-7IgA Ligand
  • Track 3-8Affibody-Fusions
  • Track 3-9Immunological Assay
  • Track 3-10Transcriptomics
  • Track 3-11Immunodiagnostic
  • Track 3-12Characterization of Lymphocytes
  • Track 3-13Recombinant Immuno-Conjugates
  • Track 3-14Microarray
  • Track 3-15Antigen-Antibody Interactions
  • Track 3-16Proteomics
  • Track 3-17Other Related Topics

 A child suffering from allergies or different issues with his insusceptible framework is eluded as Pediatric Immunology.  Child’s immune system battles against infections. On the off chance that the child has hypersensitivities, their resistant framework wrongly responds to things that are normally innocuous. Pet dander, dust, tidy, form spores, bug stings, nourishment, and solutions are cases of such things. This response may make their body react with medical issues, for example, asthma, roughage fever, hives, dermatitis (a rash), or an exceptionally extreme and unordinary response called anaphylaxis. Once in a while, if your child’s invulnerable framework is not working right, he may experience the ill effects of successive, serious, or potentially unprecedented diseases. Cases of such contaminations are sinusitis (aggravation of at least one of the sinuses), pneumonia (disease of the lung), thrush (a growth contamination in the mouth), and abscesses (accumulations of discharge encompassed by kindled tissue) that continue returning.

  • Track 4-1Primarily Genetic Diseases
  • Track 4-2Chronic Sinusitis
  • Track 4-3Immune Infertility
  • Track 4-4Children and Clinical Studies
  • Track 4-5Asthma
  • Track 4-6Preventing Disease & Life-saving Immunizations
  • Track 4-7Treatments of Pediatric Diseases
  • Track 4-8Pediatric Infectious Diseases
  • Track 4-9Pediatric Hypersensitivity
  • Track 4-10Neuropsychiatric Disorder
  • Track 4-11Acquired Immunodeficiency
  • Track 4-12Adolescent Medicine

A decline in immune competence is well recognized in the elderly. Aged people show a decline in many aspects of protective immunity including a tendency to produce lower-affinity antibodies, a failure to generate long-lasting immunity to vaccination and a loss of delayed-type hypersensitivity to antigens previously encountered in life. Bacterial and viral diseases such as tuberculosis and herpes zoster (shingles), respectively, are found much more frequently in the elderly compared to young adults. Septicemia (infectious microbes in the bloodstream) is also more common in the elderly. Pneumonia is more prevalent and more often fatal and other viral and bacterial infections are more common in older people leading to an increase in morbidity and mortality. This decline in immune competence is not solely a result of a defective immune system, as it is also a result of changes in the endocrine and nervous systems, as well as nutritional and other factors including the general state of health of the older individual.

Malignancies are seen much more frequently in older people and while many of these may be related to inappropriate DNA translational events, a defective immune system may also be responsible since there is an association between immune deficiency and increased malignancy. Defects in all compartments of the immune system have been reported in the elderly. While studies are often contradictory, reliable data indicate that defects develop in T and B cell immunity as well as in the phagocytic component of immunity. Increased NK cell numbers and decreased γδ T cell function is also a feature of aging. IL-6 and IL-10 production by monocytes is increased with aging as well as the pro-inflammatory cytokines IL-1β and TNFα. MHC molecules are expressed at a lower density on a variety of cells and fewer T cells expressing CD28, important for T cell signaling, are found in the elderly. Antibody responses are usually of lower affinity and autoantibodies are found much more frequently. Hemopoiesis is impaired with fewer progenitor cells produced. Thymic involution is well established in the elderly with fewer T cells entering the vascular pool and hence secondary lymphoid organs. AICD and apoptosis are increased. Age-related changes in hormonal and neurotransmitter function may also have an impact on immune function and may determine morbidity, mortality, and longevity.

  • Track 5-1Hemopoiesis and Aging
  • Track 5-2Thymic Involution and Aging
  • Track 5-3AICD and Apoptosis in Aging
  • Track 5-4Aging and Innate Immunity
  • Track 5-5The Effects Of Aging On Humoral Immunity
  • Track 5-6The Effects Of Aging On T-cell Immunity
  • Track 5-7Immunosenecence and Morbidity, Mortality and Longevity

Clinical Immunology has developed in the course of recent decades from a transcendent research center base to a joined clinical and lab claim to fame. The clinical work of Immunologists is to a great extent out-patient based and includes essential immunodeficiency, hypersensitivity, immune system rheumatic ailment and systemic vasculitis (mutually with Rheumatologists), joint pediatric centers for kids with immunodeficiency and sensitivity and immunoglobulin imbuement facilities for patients with immunizer lack. On the research center front, Consultant Immunologists are in charge of coordinating demonstrative immunology benefits and play out an extensive variety of obligations including clinical contact, elucidation and approval of results, quality confirmation and test improvement.

  • Track 6-1Immunological Techniques
  • Track 6-2Immunological Aspects of Endocrine Disease
  • Track 6-3Immunohematology
  • Track 6-4Basic & Advanced Clinical Immunology
  • Track 6-5HIV
  • Track 6-6Immune Deficiency
  • Track 6-7Immunological Aspects of Immunodeficiency Diseases
  • Track 6-8Immunological Aspects of Infection
  • Track 6-9Immune Regulation
  • Track 6-10Immunological Aspects of Renal Disease

Lymphoproliferative disorders are an arrangement of scatters described by the abnormal proliferation of lymphocytes into a monoclonal lymphocytosis. The two noteworthy sorts of lymphocytes are B cells and T cells, which are gotten from pluripotent hematopoietic foundational microorganisms in the bone marrow. People who have some kind of brokenness with their immune system are powerless to build up a lymphoproliferative disorder since when any of the various control purposes of the resistant framework end up noticeably useless, immunodeficiency or deregulation of lymphocytes will probably happen. There are a few acquired quality transformations that have been distinguished to bring about lymphoproliferative disorder; in any case, there are additionally gained and iatrogenic causes.

  • Track 7-1Follicular Lymphoma
  • Track 7-2Lymphoid Interstitial Pneumonia
  • Track 7-3Poems Syndrome
  • Track 7-4MCD in Immunology
  • Track 7-5Castleman Disease
  • Track 7-6Clinical Manifestations & Diagnosis
  • Track 7-7Epstein-Barr virus (EBV)
  • Track 7-8Lymphoproliferative Disorder
  • Track 7-9Wiskott-Aldrich Syndrome
  • Track 7-10Macroglobulinemia
  • Track 7-11Multiple Myeloma
  • Track 7-12T-cell lymphomas
  • Track 7-13B-cell lymphoma
  • Track 7-14Hairy Cell Leukemia
  • Track 7-15Acute Lymphoblastic Leukemia
  • Track 7-16Chronic Lymphocytic Leukemia
  • Track 7-17Other Related Topics

Immunodermatology thinks about skin as an organ of insusceptibility in wellbeing and malady. A few territories have extraordinary consideration, for example, photograph immunology (impacts of UV light on skin guard), incendiary illnesses, for example, Hidradenitis suppurativa, unfavorably susceptible contact dermatitis and atopic dermatitis, probably immune system skin ailments, for example, vitiligo and psoriasis, lastly the immunology of microbial skin ailments, for example, retrovirus contaminations and sickness. New treatments being developed for the immunomodulation of basic immunological skin sicknesses incorporate biologicals gone for killing TNF-alfa and chemokine receptor inhibitors.

  • Track 8-1Cutaneous Dendritic Cells in Health and Disease
  • Track 8-2Iatrogenic Immunodeficiency & Skin Disease
  • Track 8-3Granulomatosis
  • Track 8-4Vitiligo
  • Track 8-5Alopecia Areata
  • Track 8-6Cutaneous Lupus Erythematosus
  • Track 8-7Fibrotic Skin Diseases
  • Track 8-8Pemphigoid Spectrum
  • Track 8-9Epidermolysis Bullosa Acquisita
  • Track 8-10Immunoglobulin Dermatoses
  • Track 8-11Diagnostic procedures in Immunodermatology
  • Track 8-12Advances in Immuno-Dermatology
  • Track 8-13Topical Immune Response Modifiers
  • Track 8-14Biologic Therapies for Inflammatory Disease
  • Track 8-15Allergic Urticaria
  • Track 8-16Photoimmunology
  • Track 8-17Angiogenesis for the Clinician
  • Track 8-18Contact Dermatitis: Allergic and Irritant
  • Track 8-19Atopic Dermatitis
  • Track 8-20Immunoperoxidase Technique
  • Track 8-21Nonmelanoma Skin Cancer
  • Track 8-22Immunobiology & Immune-Based Therapies
  • Track 8-23Drug Eruptions
  • Track 8-24Immunodermatology & Viral Skin Infection
  • Track 8-25Clinical Dermatology
  • Track 8-26Cutaneous T-Cell Lymphoma
  • Track 8-27Emerging Biologics in Dermatology
Immunohematology is the study of the immunology and genetics of blood cell antigens, blood groups and specific blood proteins(such as complement) and antibodies esp. important in blood banking and transfusion medicine.
Immunohematology is more commonly known as "blood banking" or “blood biology”.
This is the field of laboratory medicine dealing with preparing blood components and blood for transfusion as well a the selection of compatible and appropriate components for transfusion. Individuals may become immunized to red blood cells due to previous exposure to red blood cells(RBCs) of other people, most commonly through transfusion or pregnancy. Our children basically receive immunizations which leads to form antibodies against the immunizing material, such as tetanus. If they are again exposed to the organism which causes tetanus, the antibodies will destroy them before they can cause infection. In a similar way, red blood cells have specific structures on their surface called antigens. If an individual is pregnant with a fetus or transfused with blood that possesses structures which the recipient or mother does not have, this may induce the individual to form antibodies. These antibodies may then destroy red blood cells(RBCs) which possess the antigen if additional sessions of transfusions are needed. That is why all blood banks will "screen" potential blood recipients for unexpected antibodies and they will then select blood which lacks the offending antigen. The formation of these antibodies is an unusual occurrence, occurring in approximately 0.1-3% of the general population.
  • Track 9-1Blood collection and processing
  • Track 9-2Autoimmune hemolytic anemia and drug-induced hemolytic anemia
  • Track 9-3Hemolytic disease of the newborn
  • Track 9-4Adverse effects of transfusion Transfusion-transmitted diseases
  • Track 9-5Transfusion therapy and the role of the medical director in blood banking
  • Track 9-6Human leukocyte antigens
  • Track 9-7Other blood group systems
  • Track 9-8The ABO & Rh blood group system
  • Track 9-9Pretransfusion testing
  • Track 9-10Molecular testing for blood groups in transfusion medicine
  • Track 9-11Red cell antibody detection and identification
  • Track 9-12Blood components: preparation, storage, and transport
  • Track 9-13Automated blood collection of blood products
  • Track 9-14Quality assurance and safety in immunohematology
Our immune system distinguishes two categories of foreign substances. One category consists of antigens (foreign substances) that are freely circulating in the body. These include molecules, viruses, and foreign cells. The Second Category consists of self-cells that display aberrant MHC(Major Histocompatibility Complex) proteins. These aberrant MHC proteins can originate from antigens that have been engulfed and broken down (exogenous antigens) or from tumor cells and virus‐infected that are actively synthesizing foreign proteins (endogenous antigens).
Depending on the kind of foreign invasion, two different immune responses occur:
The Humoral response (or antibody‐mediated response) involves B cells that pathogens or recognize antigens that are circulating in the lymph or blood (“humor” is a medieval term for body fluid). In this, the antigens bind to B cells which lead to Interleukins or helper T cells stimulate B cells. In most cases, both an antigen and a stimulator are required to activate a B cell and initiate B cell proliferation. Further, B cells proliferate and produce plasma cells. The plasma cells bear antibodies with the identical antigen specificity as the antigen receptors of the activated B cells. The antibodies are released and circulate through the body, binding to antigens. Finally, B cells produce memory cells which provide future immunity.
The Cell‐mediated response involves mostly T-cells and responds to any cell that displays aberrant MHC markers, including cells invaded by pathogens, tumor cells, or transplanted cells. Self-cells or Antigen Presenting Cells (APCs) displaying foreign antigens bind to T cells. The Interleukins (secreted by APCs or helper T cells) stimulate activation of T cells. If endogenous antigens and MHC‐I are displayed on the plasma membrane, T cells proliferate, producing cytotoxic T cells. The Cytotoxic T cells destroy cells displaying the antigens. If exogenous antigens and MHC‐II are displayed on the plasma membrane, T cells proliferate, producing helper T cells. The Helper T cells release interleukins (and other cytokines), which stimulate B cells to produce antibodies that bind to the antigens and stimulate nonspecific agents (NK and macrophages) to destroy the antigens.
  • Track 10-1T cells and B cells
  • Track 10-2Non-antibody immunity
  • Track 10-3Monocytes and Macrophages
  • Track 10-4Complement system
  • Track 10-5Inflammation and Inflammatory factors
  • Track 10-6Functions of Antibodies
  • Track 10-7Immunoglobulins
  • Track 10-8Antigen receptor sites
  • Track 10-9Adaptive immunity
  • Track 10-10Resistance to Intracellular Microbial and Viral infection

The mucosal surfaces of the body have a higher danger of contamination because of their cooperative energy with the outside condition. Mucosal immunology raises those parts of the resistant framework that shield the body from contamination. It gives three main functions secures the mucous film against disease, keeping the take-up of antigens, microorganisms, and other outside materials, and directing the creature's insusceptible reaction to that material. The mucosal insusceptible framework is contained mechanical (bodily fluid), concoction and cell elements.

  • Track 11-1Protective Mucosal Immune Responses
  • Track 11-2Influences on Mucosal Immunity
  • Track 11-3Mucosal Tolerance and Disease
  • Track 11-4Role and Regulation of IgA
  • Track 11-5Immunologically Privileged Sites
  • Track 11-6Other Related Topics
  • Track 11-7Other Related Topics
  • Track 12-1Anterior uveitis
  • Track 12-2Peripheral neuropathy
  • Track 12-3Non-caseating granuloma
  • Track 12-4Human Toxocariasis
  • Track 12-5Immunology of Ocular Tumors
  • Track 12-6Orbital Inflammation and Infection
  • Track 12-7Allergy and Ocular Surface
  • Track 12-8Immune Keratitis
  • Track 12-9Diabetes and Nonuveitic Ocular Inflammation
  • Track 12-10Epidemiology and Clinical Trials in Ocular Inflammation
  • Track 12-11Ocular Manifestations of Pathogenic Infection
  • Track 12-12Neurosarcoidosis
  • Track 12-13Retinal vasculitis

The immune system has the capacity to mount an immune response to virtually all molecules and/or cells. Although the capacity to respond to self-antigen is present in all of us, in most instances such responses result in tolerance, indicating that mechanisms must exist to prevent or subdue autoimmune responses. Moreover, auto-reactive T and B cells, as well as autoantibodies, are found in people who do not have autoimmune diseases, demonstrating that immunological auto-reactivity alone is not sufficient for the development of the disease. The mechanisms currently thought to prevent/dampen autoimmune responses include inactivation or deletion of autoreactive B and T cells, active suppression by cells or cytokines, idiotype or anti-idiotype interactions, and the immunosuppressive adrenal hormones, the glucocorticoids. When dampening mechanisms fail or are overridden, a response directed against self-antigen can occur, resulting in autoimmune diseases that range from those which are organ-specific (diabetes and thyroiditis) to those which are systemic (non-organ specific) such as systemic lupus erythematosus and rheumatoid arthritis.

  • Track 13-1Autoimmune Research: An Overview
  • Track 13-2Inflammasome
  • Track 13-3Immunometabolism & Its Regulator
  • Track 13-4Immunomodulatory Effects on Immune System
  • Track 13-5Immunotherapy: powerful-but-Blunt Weapons
  • Track 13-6Radio Immunotherapy & Cancer vaccines Development
  • Track 13-7Current Evidence and Future Perspectives of Autoimmunity
  • Track 13-8Future Immunomodulation Strategies
  • Track 13-9Autoimmunity: A Look at Interesting New Research
  • Track 13-10Immunomodulatory Xenobiotics

Reproductive immunology refers to a field of medicine that studies interactions (or the absence of them) between the immune system and components related to the reproductive system, such as maternal immune tolerance towards the fetus, or immunological interactions across the blood-testis barrier. The immune system refers to all parts of the body that work to defend it against harmful enemies. In people with immunological fertility problems, their body identifies the part of reproductive function as an enemy and sends Natural Killer (NK) cells to attack. A healthy immune response would only identify an enemy correctly and attack only foreign invaders such as a virus, parasite, bacteria, etc.

The concept of reproductive immunology is not widely accepted by all physicians. Those patients who have had repeated miscarriages and multiple failed IVF's find themselves exploring its possibilities as the reason. With an increased amount of success among treating any potential immunological factors, the idea of reproductive immunology can no longer be overlooked. The failure to conceive is often due to immunologic problems that can lead to very early rejection of the embryo, often before the pregnancy can be detected by even the most sensitive tests. Women can often produce perfectly healthy embryos that are lost through repeated "mini miscarriages." This most commonly occurs in women who have conditions such as endometriosis, an under-active thyroid gland or in cases of so-called "unexplained infertility." It has been estimated that an immune factor may be involved in up to 20% of couples with otherwise unexplained infertility. These are all conditions where abnormalities of the woman’s immune system may play an important role.

  • Track 14-1Current trends in reproductive immunology
  • Track 14-2Endometriosis
  • Track 14-3Clinical practices in reproductive immunology
  • Track 14-4Immuno-contraceptive Vaccine
  • Track 14-5Sexually Transmitted Infections & Diseases
  • Track 14-6Immune-mediated sub-fertility and pregnancy complications
  • Track 14-7Immunogenetics in reproduction
  • Track 14-8Immunoregulatory Responses

Microbial immunology is the investigation of the molecular mechanisms utilized by microorganisms to bring about sickness in people and creatures. Bacterial, protozoan, parasitic and viral pathogens have built up a wide assortment of devices to build up themselves in the host and pick up supplements, which additionally cause impedance and malady. To comprehend the mind-boggling forms utilized by microbial pathogens, microbiologists and immunologists utilize every one of the devices of present-day sub-atomic science, hereditary qualities, harmfulness elements, tranquilize collaborations, organic chemistry, and biophysics. Seeing how microorganisms cause ailment is regularly the initial move toward the improvement of new antibodies and therapeutics and its cover all parts of the interrelationship between irresistible specialists and their hosts.

  • Track 15-1Cellular Responses to Bacterial, Parasitic, Viral and Fungal Pathogens
  • Track 15-2B-Glucan & Anti-Fungal Immunity Prion
  • Track 15-3Hepatitis Viruses
  • Track 15-4Apoptosis & Viral Infection
  • Track 15-5Helminth Parasites
  • Track 15-6Mechanisms of Host Invasion, Evasion, and Resistance of Virus (including HIV)
  • Track 15-7Mechanisms of Host Invasion, Evasion, and Resistance
  • Track 15-8Innate Immunity Against Bacterial, Parasitic, Viral and Fungal Pathogens
  • Track 15-9Immunopathogenesis of Bacterial, Parasitic,Viral and Fungal Infection
  • Track 15-10Other Related Topics

Nutrition is critical to immune defense and resistance to pathogens, with consequences that affect the health, welfare, and reproductive success of individual organisms, and also has profound ecological and evolutionary implications. In humans, under-nutrition or under-nourished, notably of protein, is a major contributor to mortality and morbidity due to infectious diseases, particularly in the developing world. Likewise, over-nutrition and its associated metabolic disorders may impair immune function, increase susceptibility to infectious disease, and disrupt the relationship with symbiotic and commensal microbiota.

The Nutritional Immunology investigates the role of dietary components and their interactions with other environmental factors and genes in age-associated changes of the immune and inflammatory responses. Various researches are going on to look on to determine the molecular mechanisms by which nutrients modulate immune cell functions. Methods are being developed to use the immune response as a biologically meaningful index in determining specific dietary requirements and reverse and/or delay the onset of these immunologic and age-related changes by appropriate dietary modifications.

The new era of research approach is to: 1) Study molecular and cellular mechanisms of age and nutrition-induced changes in immune and inflammatory responses.

2). Determine the efficacy of food components such as micronutrients and phytochemicals in the prevention of infectious, autoimmune and chronic diseases in animal models, clinical trials and observational studies in developed and less developed countries.

3) Determine the efficacy of food components including total calories, lipids, micronutrients such as vitamin E, and zinc, as well as flavonoids such as those in green tea and wolfberry, and pre- and pro-biotics on improving the immune function and/or dampening the inflammatory responses using cell culture, animal models, and clinical trials

4) Investigate the impact of nutrition during fetal life on long-life resistance to immune and inflammatory diseases.

5) Determine the life-long impact of obesity and reducing caloric intake on immune response and resistance to infection.

  • Track 16-1Bio active Nutrients
  • Track 16-2Dietary Metabolic
  • Track 16-3Animal Nutrition Maternal and Infant Nutrition
  • Track 16-4Animal Nutrition
  • Track 16-5Public Health Nutrition
  • Track 16-6White-Brown Adipocyte Plasticity & Inflammation
  • Track 16-7Early life Nutritional Influences on the Immune System
  • Track 16-8Nutrient-Gene Interaction in the Immune System
  • Track 16-9Obesity & Hormonal Imbalance
  • Track 16-10Eco nutrition

Eco-immunology is an explicit example of how fruitful integrative & innovative approaches to biology can be. Since its emergence, Eco -immunology has sparked constructive debate on a wide range of topics, from the molecular & cellular mechanics of immune responses to the role of immunity in shaping the evolution of life histories. To complement the symposium Methods and Mechanisms in Eco-immunology and commemorate the inception of Disease Ecology and the Division of Eco-immunology within the Society for Integrative and Comparative Biology, we appraise the origins of Eco-immunology, with a focus on its continuing and valuable integration with disease ecology. The greatest contribution of Eco-immunology to wider biology has been the establishment of immunity as an integral part of organismal biology, one that may be regulated to maximize fitness in the context of costs, constraints, and complex interactions. As we discuss historical impediments and ongoing progress in Eco-immunology, in particular, the thorny issue of what Eco-immunologists should, should not, or cannot measure, and what novel contributions Eco-immunologists have made to the understanding of host-parasite interactions. Finally, we highlight & focus some areas to which Eco-immunology are likely to contribute in the near future.

  • Track 17-1Competitive ability
  • Track 17-2Immune Regulation
  • Track 17-3Eco-immunological approach
  • Track 17-4Invasive species
  • Track 17-5Inflammation
  • Track 17-6Host–parasite coevolution
  • Track 17-7Expansion range
  • Track 17-8Co-infection
  • Track 17-9Costly immune defenses & Tolerance
  • Track 17-10Immune defense & Bio-invasion Relationship
  • Track 17-11Antigen-diversity

Organ transplantation is the moving of an organ from one body to another or from a donor site to another location on the patient's own body, for the purpose of replacing the recipient's damaged or absent organ. Transplantation immunology is the sequence of immunological reactions that occur after the transplant. A major limitation to the success of transplantation is the immune response of the recipient to the donor tissue. A major barrier to transplantation is the process of rejection, in which the recipient's immune system recognizes the graft as being foreign and attacks it. During fetal life, within the thymus, a very important process called thymic education occurs. Immune cells that react with “self” antigens are killed by a process called apoptosis. The removal of these self-reactive cells, a process called negative selection, results in tolerance to our own proteins, ie, self-tolerance, and prevents autoimmune reactions.

  • Track 18-1Bone Marrow Transplantation
  • Track 18-2Biological clock
  • Track 18-3Potential xenotransplantation
  • Track 18-4Anti-donor Antibodies & Current Research
  • Track 18-5Tolerance induction; xenotransplantation; islet cell transplantation
  • Track 18-6Chronic Rejection
  • Track 18-7Graft vs Host Disease
  • Track 18-8Hyperacute Rejection
  • Track 18-9Solid Organs Transplantation
  • Track 18-10Transplantation Tolerance
  • Track 18-11Stem Cell Transplantation
  • Track 18-12Other Related Topics

Computational Immunology and Immunological Bioinformatics are firm and rapidly developing examination fields. Though the previous expects to create numerical and additionally computational strategies to concentrate the progression of the cell and atomic substances amid the resistant reaction, the last concentrates on proposing techniques to examine enormous Genomic & proteomic immunological-related datasets and foresee new information for the most part by measurable derivation and machine learning calculations. The overabundance of information delivered by high-throughput instrumentation, remarkably genomicsTranscriptomics, Epigenetics, and Proteomics techniques, requires computational apparatuses for securing, stockpiling, and investigation of immunological information. The misuse of such an enormous measure of immunological information, for the most part, requires its change into computational issues, their answer utilizing scientific and computational methodologies, and after that, the interpretation of the got comes about into immunologically important translations.

  • Track 19-1Fundamentals of Computational Immunology
  • Track 19-2Computational Modeling
  • Track 19-3Use of Computational Modeling in Immunological Research
  • Track 19-4Use of Computational Modeling in Immunological Research
  • Track 19-5Immunoinformatics Cyberinfrastructure for Modeling and Analytics
  • Track 19-6Database & Tools in Immunology
  • Track 19-7Agent-Based Modeling and High Performance Computing
  • Track 19-8From Big Data Analytics and Network Inference to Systems Modeling
  • Track 19-9Multiscale Modeling: Concepts, Technologies, and Use Cases in Immunology
  • Track 20-1Combined Immunodeficiencies
  • Track 20-2Autoinflammatory disorders
  • Track 20-3Defects in Innate Immunity
  • Track 20-4Congenital defects of phagocyte number, function, or both
  • Track 20-5Predisposing Disease
  • Track 20-6Opportunistic Infection
  • Track 20-7Complement Deficiencies
  • Track 20-8Secondary Immunodeficiency
  • Track 20-9Predominantly Antibody Deficiencies.
  • Track 20-10Combined Immunodeficiencies with Associated or Syndromic features
  • Track 20-11Immune Dysregulation.

The vaccine is a biological preparation that enhances resistance to specific infection. It contains particular operators that look like an illness bringing about the microorganism as well as animates body's safe framework to perceive the outside specialists. A vaccine is dead or inactivated life forms or refined items gotten from them. Entire creature antibodies cleaned macromolecules as vaccines, recombinant antibodies, DNA antibodies. The insusceptible framework perceives vaccine specialists as remote, devastates them, and "recollects that" them.

Immunotherapy is a standout amongst the most energizing ranges of new disclosures and medicines for various sorts of a tumor. Seeing how the insusceptible framework functions are opening the ways to grow new medicines that are changing the way we consider and treat growth. The vast majority accepting immunotherapies are dealt with in particular malignancy focuses and a large portion of them are selected in clinical trials. This may change as more trials are finished and more medications are affirmed by the Food and Drug Administration (FDA) to treat various types of malignancy.

  • Track 21-1Passive / Active Immunotherapy
  • Track 21-2Vaccines for Infectious Diseases
  • Track 21-3DNA vaccines
  • Track 21-4Cancer Vaccines
  • Track 21-5Vaccine Research & Development
  • Track 21-6Therapy of Asthma and Hypersensitivity Responses
  • Track 21-7Tumor Vaccine Development and Immune-Based Therapies
  • Track 21-8Immunotherapeutic Strategies Against Pathogens
  • Track 21-9Mucosal Vaccine Development and Immune-Based Therapies
  • Track 21-10Children vaccines
  • Track 21-11Immunotherapy Challenges
  • Track 21-12Therapeutic Modulation of Tolerance and Autoimmune Disease in Animal Models
  • Track 21-13Immune System Checkpoints
  • Track 21-14Issues in Immunotherapy
  • Track 21-15Monoclonal Antibodies
  • Track 21-16Immuno memory
  • Track 21-17Vaccination
  • Track 21-18Dendritic Cell based Immunotherapy
  • Track 21-19Cytokine based cancer therapy
  • Track 21-20Development of Therapies for Human Autoimmune Disease
  • Track 21-21Other Related Topics

An Immunological Clinical trial is a unique research program designed to cultivate a collaborative environment that allows basic and translational immunologists to work side-by-side with clinicians. Clinical trial & Practices are always designed to gain some knowledge about something not yet well-known or proven. A person may volunteer to participate in a clinical study. A physician may recommend a patient to consider volunteering for specific study participation, as part of the patient therapeutic treatment options.  Clinical trials are highly regulated and are conducted following strict scientific standards in order to protect patients and to produce meaningful results.

The clinical trial allows accelerating the bench-to-bedside transition of innovative immunotherapies, with much attention given to critical diseases which are known to relapse or is refractive to conventional treatments currently available. Many of the novel immunotherapy approaches, which originated from basic science research by the clinical trial and practices, are now being explored as new treatment modalities in patients, with a significant number advancing through clinical trials towards FDA approval.

  • Track 22-1Drug Development
  • Track 22-2Trends & Perspectives in Clinical Research
  • Track 22-3Real world Clinical Trial Strategies and Results
  • Track 22-4Challenges in Drug Discovery & Development
  • Track 22-5Drug discovery & Development in Preclinical Research.
  • Track 22-6Phases of Trials
  • Track 22-7Immunobiology
  • Track 22-8Immunohistochemistry
  • Track 22-9Immunocytochemistry
  • Track 22-10Pre-clinical Trial & Research Considerations
  • Track 22-11Clinical trials for Rare Diseases: Challenges and Opportunities

The immune system is the body's initial line of barrier against most illnesses and irresistible intruders. Seeing how the immune system functions—or does not work—against growth is an essential concentration of the Cancer Immunology Program. Specialists are decoding the fundamental science of safe reactions with the objective of growing new safe treatment approaches and enhancing existing ones. The Huge concentrate is on consolidated methodologies that expand upon disclosures in malignancy hereditary qualities (changes to DNA), epigenetics (concoction modifications to the earth of DNA) and immunology. Late revelations utilize new discoveries about how the immune system capacities to make collaboration and enhance the viability of surgery, disease antibodies, medicate treatments and radiation treatment. Accuracy, or customized, medication is utilized to control the best invulnerable ways to deal with the correct patients, and this energizing work is prompting dependable reactions in numerous growths. Eventually, scientists hope to unravel why insusceptible treatments work for a few patients and not others, enrolling the body's own particular safeguards against for all intents and purposes each sort of growth.

  • Track 23-1Carcinogenesis
  • Track 23-2Psoriasis
  • Track 23-3Other Related Topics
  • Track 23-4Immuno-Oncology studies
  • Track 23-5Economic Impact on Cancer
  • Track 23-6Specific / Non-specific Immunotherapies
  • Track 23-7Treatment Approaches for Cancer
  • Track 23-8Targeted therapies
  • Track 23-9Drug Development & Vaccines
  • Track 23-10Chemotherapy & Radiotherapy
  • Track 23-11Pathobiology of Immune System Malignancies
  • Track 23-12Mechanisms of Tumor Rejection and Modulation of Antitumor Responses
  • Track 23-13Tumor-Associated Immunosuppression
  • Track 23-14Tumor-Associated Antigens
  • Track 23-15Antitumor Effector Cells and Regulation of Tumor Immunity
  • Track 23-16Cancer therapeutic resistance
  • Track 23-17Cancer Immunology & Immunotherapy
  • Track 23-18Oncogenomics
  • Track 23-19Randomized Controlled Trial

Allergies represent TYPE I responses as per the Gell and Coombs grouping. Most are brought about by IgE ANTIBODIES which are fit for the official to Fc-receptors for IgE on tissue MAST CELLS. Cross-connecting of these layers bound IgE's by particular antigen brings about pole cell DEGRANULATION; this procedure discharges HISTAMINE and an assortment of other effector molecules, which thus brings about the bunch side effects of sensitivity. (rash, feed fever, asthma and so on.) Passive cutaneous hypersensitivity (PCA) in the guinea pig and the Prausnitz-Küstner (P-K) skin response in people give models to understanding the basic component of unfavorably susceptible responses. Administration of sensitivities starts with allergen shirking and incorporates the utilization of an assortment of medications and allergen-particular DESENSITIZATION.

  • Track 24-1The production of IgE
  • Track 24-2Market analysis of Allergy & Hypersensitivity
  • Track 24-3Asthma and Allergic Inflammation
  • Track 24-4Haptens
  • Track 24-5Adaptive (T & B Cell) Immune Responses
  • Track 24-6Mechanisms of Hypersensitivity with Mediators & Examples
  • Track 24-7Allergy Prevention, Risk Factors & Treatment
  • Track 24-8Allergy Diagnosis & Medicine
  • Track 24-9Effector mechanisms in Allergic Reactions
  • Track 24-10Types of Allergies
  • Track 24-11Mast Cells, Eosinophils, Basophils
  • Track 24-12Types of Hypersensitivity & Immuno Complexes
  • Track 24-13Other Related Topics

The investigation of the connection between our  central nervous system (the brain and spinal string) and our immune system. Neuroimmunology adds to advancement of new pharmacological medications for a few neurological conditions. The immune system and the sensory system keep up broad correspondence, including "hardwiring" of thoughtful and parasympathetic nerves to lymphoid organs. Neurotransmitters, for example, acetylcholine, norepinephrine, vasoactive intestinal peptide, substance P and histamine adjust the insusceptible action. Neuroendocrine hormones, for example, corticotrophin-discharging element, leptin, and alpha-melanocyte empowering hormone manage cytokine adjust. The immune system adjusts brain action, including body temperature, rest and bolstering conducts. Particles, for example, the significant histocompatibility complex not just direct T cells to immunogenic molecules held in its separated additionally regulate improvement of neuronal associations. Neurobiologists and immunologists are investigating normal thoughts like the neurotransmitter to comprehend properties.

  • Track 25-1Neuroimmune Interaction
  • Track 25-2Neuroinflammatory Disorders
  • Track 25-3Neural stem cell transplantation
  • Track 25-4Neuro immuno endocrine system
  • Track 25-5Interferon signaling and neuro immunology
  • Track 25-6Experimental neuro immunology
  • Track 25-7Immune-Mediated Neurological Syndromes

Every living creature is constantly presented to substances that are not fit for their upbringing. Most living beings secure themselves against such substances in more than one way with physical barriers, for example, or with chemicals that repulse or slaughter invaders. Creatures with spines, called vertebrates, have these sorts of general defensive instruments; however, they additionally have a more progressed defensive framework called the immune system. The invulnerable framework is a perplexing system of organs containing cells that perceive outside substances in the body and devastate them. It secures vertebrates against pathogens, or irresistible specialists, for example, infections, microscopic organisms, growths, and different parasites. The human immune system is the most complex. Although there are numerous possibly unsafe pathogens, no pathogen can invade or attack all organisms because a pathogen's ability to cause harm requires a susceptible victim, and not all creatures are powerless to similar pathogens. For example, the infection that causes AIDS in people does not contaminate creatures, for example, dogs, cats, and mice. Correspondingly, people are not defenseless to the infections that cause canine distemper, cat leukemia, and mousepox.

  • Track 26-1Cardiac immunopathology
  • Track 26-2Differentiation and Selection
  • Track 26-3Hematopoiesis and Development
  • Track 26-4Evolution and Genetic Regulation of the Immune System
  • Track 26-5Comparative Immune Responses to Pathogens/Tumors
  • Track 26-6Abnormalities in Immune System
  • Track 26-7Super antigens: staphylococcus and streptococcus
  • Track 26-8Immunotherapy Challenges
  • Track 26-9Haemorrhagic fevers & pathophysiology
  • Track 26-10Scleroderma
  • Track 26-11Respiratory immunopathology
  • Track 26-12Neurological immunopathology
  • Track 26-13Other Related Topics